Perhaps you can explain the following then:
- The drawback phenemenon is not usually observed with. 22 rifles and low velocity ammo.
- A silencer makes it less likely still.
- When it is observed it usually involves contact gunshot wounds to the head
- When it is observed it usually includes tissue with the blood
- No comparable cases in criminal history where blood inside a silencer underpins a conviction.
- None of the forensic literature, research, tests include drawback with a silencer
- No tests/modelling have been undertaken to establish whether the drawback phenemenon is possible with the rifle, ammo and silencer.
- The blood flake inside the silencer was the only blood stained exhibit capable of yielding the results claimed.
- The non-porous blood stained rifle was unable to yield any results beyond human in origin
- Blood analysed by serology requires good quality samples of a certain quantity. Heat and humidity are known to degrade samples rendering them useless for serological analysis. The blood flake had to withstand heat from firearm discharge, humidity in the cyanoacrylate fuming chamber and gunshot residue.
- Victims' blood samples were analysed for:
- Antigens - ABO
- Haptoglobin - HP - Protein
- Phosphoglucomutase - PGM - Enzyme
- Adenylate Kinase - AK - Enzyme
- Erythrocyte Acid Phosphatase - EAP - Enzyme
The samples yielded results for all the above.
The blood flake in the silencer was analysed but unable to yield a result for the enzyme PGM. This contradicts advice. I was given by the Chief Forensic Serologist at the Serological Research Institute in Californis that PGM is more stable than AK and EAP which begs the question how/why in this case the flake was unable to yield a result for PGM? This is currently being reviewed by a forensic scientist in private practice along with other 'evidence'.
Malcolm Fletcher wasn't giving his opinion of whether drawback would cause blood to enter the silencer of
any gun, but based his opinion on the
location of the blood found and the expert evidence of who did or might have received contact wounds that matched the blood types found.
Other methods of blood entering the moderator were discussed including a bloody area being struck at force and blood simply dripping or dropping into the end. Neither were completely ruled out but it was MFs opinion that if this had occurred, blood would not have travelled as far as the 5th and possibly 7th baffle.
The bloodflake was only a quarter of an inch in size, perhaps it was too small for 1985 serology to detect PGM no matter how stable it was. No PGM was detected in any other samples but AK was. You actually state in your own blood tables post that PGM
"Breaks down quickly outside the body hence blood in silencer was unable to produce a reading" so which is it? Is PGM more stable than AK and is The Chief Forensic Serologist at the Serological Research Institute talking about 1985 methods used in serology or more modern ones that would be better at detecting PGM?